Injury to mitochondria — tiny powerhouses inside cells that provide them with energy — triggers events that occur early in Alzheimer’s disease and may offer a promising target for treatment. So concludes a new study published in the journal Alzheimer’s & Dementia that shows how oligomeric amyloid beta —a highly toxic protein — disrupts mitochondria. The study also reveals how a pre-treatment might protect human brain cells from such damage.
Alzheimer’s: Protecting ‘powerhouse’ of cells may fuel new treatment
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